Cerebellar Lesions :: Neurology Medical Health Essays

Cerebellar Lesions The cerebellum integrates sensory and other inputs to coordinate ongoing movements and recruit in motor planning. The cerebellum has no direct connections to the begin motor neurons still modulates motor kick the bucket through upper motor neurons. The Vermis and flocculonodular lobes act upon balance and eye movements via connections with vestibular nuclei and oculomotor system, these regions control the medial motor systems. The more lateral areas of the cerebellum control muscular tissues of extremities, the most lateral areas control function in motor planning. (Chapter 15 Cerebellum) When cerebellar lesions occur they produce dyssynergia (irregular uncoordinated movement.) Ataxia occurs ipsilateral to the typeface of the cerebellar injury. Midline cerebellar lesions cause unsteady gate (truncal ataxia) and eye movement abnormalities (nystagymas), which are frequently accompanied by vertigo, nausea and vomiting. Lateral cerebellar lesions cause weapon system ataxia. (Chapter 15 Cerebellum) Patients with cerebellar damage, regardless of the cause or location, exhibit forbidding errors in movement. These movement errors are always on the same side of the body as the damage to the cerebellum, reflecting the cerebellums unusual status as a brain structure in which sensory and motor information is represent ipsilateral rather than contra laterally. Furthermore, somatic, visual, and other inputs are represented topographicly in spite of appearance the cerebellum as a result, the movement deficits may be quite specific. For example, virtuoso of the most common cerebellar syndromes is caused by degeneration in the foregoing portion of the cerebellar cortex in patients with a long accounting of alcohol abuse. Such damage specifically affects movement in the lower limbs, which are represented in the anterior spinocerebellum. The consequences include a ample and staggering gait, with little impairment of arm or h and movements. Thus, the topographical organization of the cerebellum allows cerebellar damage to disrupt the coordination of movements performed by some muscle groups but not others. The implication of these pathologies is that the cerebellum is normally capable of integrating the moment-to-moment actions of muscles and joints end-to-end the body to ensure the smooth execution of a full betray of motor behaviors. Thus, cerebellar lesions lead first and foremost to a inadequacy of coordination of ongoing movements. For example, damage to the vestibulocerebellum impairs the ability to stand upright and maintain the snap of gaze. The eyes have difficulty